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Case Study Cancer

Mr. L. is a 69 year-old male with acute myelogenous leukemia (AML), FAB-M1 normal cytogenetics diagnosed in May of 2001. He received chemotherapy (7+3: AraC/idarubican) at that time and achieved complete remission. He developed a relapse in the April of 2002. He deferred treatment as he was trying alternative medicines. He eventually was treated with chemotherapy (Mylotarg) again in May of 2002, but this did not result in remission and he is considered to be in refractory relapse.

Mr. L. has a history of neutropenic fever, otitis media, diabetes, positive VRE in his blood, and Coronary Artery Disease. Mr. L does understand that there is no cure for his disease and that is why he has chose alternative care. He has refused a referral to UW-Madison & UI-Chicago for research and experimental therapy.

At this time he is still trying alternative medicines, and comes into the Cancer Center for lab draws, and if needed he has blood and/or platelet transfusions. He has refused Procrit and GM-CSF, to assist in his condition.

Mr. L is married and his wife is supportive of him and his choice in attempting alternative medicine. He also has many siblings who are supportive of him and 2 of them were t


Mr. L. is in Erikson’s stage of generativity versus stagnation. I feel that he is in the generativity part of this stage. Generativity is where the person is able to assist the younger generation in developing and leading useful lives. He is doing this by giving back to his family and being their for them and trying to go on in his normal routine.

The subclassification of the disease is important. Different types of therapy may be used and the likely course of the disease may be different. Additional features may be important in guiding the choice of therapy, including: abnormalities of chromosomes, the cell immunophenotype, the age and the general health of the patient, and others.

Older people are more likely to develop the disease. AML incidence increases dramatically among people who are over the age of 40. They are most prevalent in the sixth, seventh and eighth decades of life.

AML results from acquired (not inherited) genetic damage to the DNA of developing cells in the bone marrow. The effects are: 1) the uncontrolled, exaggerated growth and accumulation of cells called "leukemic blasts" which fail to function as normal blood cells and 2) the blockade of the production of normal marrow cells, leading to a deficiency of red cells (anemia), and platelets (thrombocytopenia) and normal white cells (especially neutrophils, i.e., neutropenia) in the blood.

Some topics in this essay:
Lab Values, Leukemia Leukemia, PLT CNT, AML AML, AML FAB-M1, Procrit GM-CSF, CBC CMP, NEUT ABS, Risk Factors, MONO ABS, blood cells, bone marrow, white blood, myelogenous leukemia, red blood cells, white blood cells, red blood, cells bone marrow, cells bone, acute myelogenous, acute myelogenous leukemia, trying alternative, blood red, alternative medicines, trying alternative medicines,

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Approximate Word count = 1113
Approximate Pages = 4 (250 words per page double spaced)


  

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