To understand CBW, one must first appreciate the method of attack these chemical agents are capable of. For this project, we were asked to investigate weapons that pursued neuronal means of assault, affecting neurotransmitter pathways of victims. VX gas, Sarin, and Tabun were three examples of toxins given to us that do this in their own specific ways. I explored the effects of these fatal substances both individually and collectively at http://www.sis.nlm.nih.gov/Tox/ChemWar.html, a site within the national library of medicine. All three of these nerve agents are cholinesterase inhibitors, meaning they stop the function nerve cells by blocking there impulse transmissions. This can be better explained with an analogy.

A couple new concepts that related to class were the drug effects, and the psychological consequence of chemical warfare. The nerve agents described previously take direct action on the nerve impulse process that we are learning about in this class. The psychological warfare aspects relate to our school work in that they encompass the mind’s probable perspective of possible CBW attack.

Imagine the nerve cell like a giant arm extending from the body. At the end of this arm is a hand with long fingers that release chemical messages out the end of their fingertips. These messages are released to and received by a closely linked person whom the arm is outstretched towards. The recipient nerve, or in this case person, has special receptors on its head so that it can properly receive the message sent from the original person. Neurotoxins act to neutralize the chemicals that allow those special receptors to do their job correctly. In other words, it is like a shampoo is put on the person’s head that binds not to the receptors, but to the chemicals that allow the receptors to work properly. The receptors are flooded with excessive acetylcholine and subsequent damage follows. In this way neurotoxins disable neurons from operating normally. The site I mentioned earlier clarifies the effectiveness of this kind of neuronal malfuncti

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