However, Stricker and Zigmond (1976), reviewed the evidence and concluded that behvioural effects (catatonic behaviour of rats) of lateral hypothalamic lesions, including eating suppression, were produced by damage to axons that pass through the region, known to play a role in the control of movement. Also injections activate two populations of neurons located in the lateral hypothalamus that stimulate hunger and decrease metabolic rate. The neurons secrete two different peptide neu

Neurons that secrete NPY are found in the arcuate nucleus, located in the hypothalamus at the base of the third ventricle. The nucleus also contains neurosecretory cells whose hormones control the secretions of the anterior pituitary gland. The NPY neurons send a projection of axons to the paraventricular nucleus, the region where infusions of NPY affect metabolic functions. They also send a projection directly to MCH and orexin neurons in the lateral hypothalamus. Presumably these connections are responsible for the feeding elicited by activation of NPY neurons (Carlson, 2001, 410).

Researchers have not yet determined by what pathways NPY neurons in the arcuate nucleus receive information concerning the animal’s nutritional status. (Carlson, 2001, 410). These neurons receive input from a variety of forebrain and hindbrain structures, so it is possible that these connections convey information from detectors that are sensitive to glucoprivation and lipoprivation (Li, Chen and Smith, 1999).

Some topics in this essay:
Stricker Zigmond, Chen Smith, NPY Neurons, VMH PVN, Chang McDonald, Kalra Kalra, Flood Morley, , Infusion NPY, Anand Brobeck, lateral hypothalamus, mch orexin, npy neurons, orexin neurons, mch orexin neurons, insulin secretion, neurons lateral hypothalamus, arcuate nucleus, paraventricular nucleus, vmh lesions, ventromedial hypothalamus, et al, orexin neurons lateral, autonomic nervous system, control autonomic nervous,